PENDIN, DIANA

PENDIN, DIANA  

Istituto di Neuroscienze - IN -  

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Risultati 1 - 20 di 26 (tempo di esecuzione: 0.004 secondi).
Titolo Data di pubblicazione Autore(i) File
A Synthetic Fluorescent Mitochondria-Targeted Sensor for Ratiometric Imaging of Calcium in Live Cells 1-gen-2019 Pendin, D; Norante, R; De Nadai, A; Gherardi, G; Vajente, N; Basso, E; Kaludercic, N; Mammucari, C; Paradisi, C; Pozzan, T; Mattarei, A
Balancing ER dynamics: Shaping, bending, severing, and mending membranes 1-gen-2011 Pendin, Diana; McNew James, A; Daga, Andrea
Ca2+ and cAMP cross-talk in mitochondria 1-gen-2014 Di Benedetto, Giulietta; Pendin, Diana; Greotti, Elisa; Pizzo, Paola; Pozzan, Tullio
Calcium Imaging in Drosophila melanogaster 1-gen-2020 Vajente N.; Norante R.; Pizzo P.; Pendin D.
Characterization of the ER-targeted low affinity Ca2+ probe D4ER 1-gen-2016 Greotti E.; Wong A.; Pozzan T.; Pendin D.; Pizzo P.
Dynamic constriction and fission of endoplasmic reticulum membranes by reticulon 1-gen-2019 Espadas, Javier; Pendin, Diana; Bocanegra, Rebeca; Escalada, Artur; Misticoni, Giulia; Trevisan, Tatiana; Velasco del Olmo, Ariana; Montagna, Aldo; Bova, Sergio; Ibarra, Borja; Kuzmin Peter, I; Bashkirov Pavel, V; Shnyrova Anna, V; Frolov Vadim, A; Daga, Andrea
Exploring cells with targeted biosensors 1-gen-2016 Pendin, D; Greotti, E; Lefkimmiatis, K; Pozzan, T
Familial Alzheimer's disease presenilin-2 mutants affect Ca2+ homeostasis and brain network excitability 1-gen-2019 Pendin D.; Fasolato C.; Basso E.; Filadi R.; Greotti E.; Galla L.; Gomiero C.; Leparulo A.; Redolfi N.; Scremin E.; Vajente N.; Pozzan T.; Pizzo P.
Familial Alzheimer's disease-linked presenilin mutants and intracellular Ca 2+ handling: A single-organelle, FRET-based analysis 1-gen-2019 Greotti, E; Capitanio, P; Wong, A; Pozzan, T; Pizzo, P; Pendin, D
Generation and Characterization of a New FRET-Based Ca 2+ Sensor Targeted to the Nucleus 1-gen-2021 Galla, Luisa; Vajente, Nicola; Pendin, Diana; Pizzo, Paola; Pozzan, Tullio; Greotti, Elisa
GTP-dependent packing of a three-helix bundle is required for atlastin-mediated fusion 1-gen-2011 Pendin, Diana; Tosetto, Jessica; Moss Tyler, J; Andreazza, Camilla; Moro, Stefano; McNew James, A; Daga, Andrea; Daga, Andrea
Homotypic fusion of ER membranes requires the dynamin-like GTPase Atlastin 1-gen-2009 Orso, Genny; Pendin, Diana; Liu, Song; Tosetto, Jessica; Tosetto, Jessica; Moss, Tyler J.; Faust, Joseph E.; Micaroni, Massimo; Egorova, Anastasia; Martinuzzi, Andrea; McNew, James A.; Daga, Andrea; Daga, Andrea; Daga, Andrea
In vivo Analysis of CRISPR/Cas9 Induced Atlastin Pathological Mutations in Drosophila 1-gen-2020 Montagna, Aldo; Vajente, Nicola; Pendin, Diana; Daga, Andrea
Manipulation of Mitochondria Dynamics Reveals Separate Roles for Form and Function in Mitochondria Distribution 1-gen-2018 Trevisan, Tatiana; Pendin, Diana; Montagna, Aldo; Bova, Sergio; Ghelli, Anna Maria; Daga, Andrea
mCerulean3-Based Cameleon Sensor to Explore Mitochondrial Ca<sup>2+</sup> Dynamics In Vivo 1-gen-2019 Greotti, E.; Fortunati, I.; Pendin, D.; Ferrante, C.; Galla, L.; Zentilin, L.; Giacca, M.; Kaludercic, N.; Di Sante, M.; Mariotti, L.; Lia, A.; GómezGonzalo, M.; Sessolo, M.; Carmignoto, G.; Bozio, R.; Pozzan, T.
Microtubules Stabilization by Mutant Spastin Affects ER Morphology and Ca2+ Handling 1-gen-2019 Vajente, N; Norante, R; Redolfi, N; Daga, A; Pizzo, P; Pendin, D
Mitofusin 2: From functions to disease 1-gen-2018 Filadi R.; Pendin D.; Pizzo P.
Optogenetic control of mitochondrial metabolism & Ca2+ signaling by mitochondria-Targeted opsins 1-gen-2017 Tkatch, T; Greotti, E; Baranauskas, G; Pendin, D; Roy, S; Nita, Li; Wettmarshausen, J; Prigge, M; Yizhar, O; Shirihai, Os; Fishman, D; Hershfinkel, M; Fleidervish, Ia; Perocchi, F; Pozzan, T; Sekler, I
Presenilin-2 and Calcium Handling: Molecules, Organelles, Cells and Brain Networks 1-gen-2020 Pizzo, Paola; Basso, Emy; Filadi, Riccardo; Greotti, Elisa; Leparulo, Alessandro; Pendin, Diana; Redolfi, Nelly; Rossini, Michela; Vajente, Nicola; Pozzan, Tullio; Fasolato, Cristina
Proteasome dysfunction induces excessive proteome instability and loss of mitostasis that can be mitigated by enhancing mitochondrial fusion or autophagy 1-gen-2019 Tsakiri Eleni, N; Gumeni, Sentiljana; Vougas, Konstantinos; Pendin, Diana; Papassideri, Issidora; Daga, Andrea; Gorgoulis, Vassilis; Juhasz, Gabor; Scorrano, Luca; Trougakos Ioannis, P